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These are most common during dose escalation and typically resolve as your body adjusts

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Aβ-42 aggregates and combines to Ca 2+ channels and AMPA (α-amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid) receptors, to which the neurotransmitter glutamate binds, so Aβ is the most neurotoxic form (3, 31)